Insulin-like Growth Factor (IGF-1)

Views 1392
Download the GMI DOC on this Topic

Sign in to Purchase/Download Documents

Sign in / Register

Focus Your Research

Click any topic below to filter + focus research
5 Diseases Researched for Insulin-like Growth Factor (IGF-1)
NameACCKFocus
Amyotrophic Lateral Sclerosis47Locked
Motor Neuron Disease36Locked
Muscular Dystrophies12Locked
Peripheral Neuropathies12Locked
Excitotoxicity 11Locked
3 Pharmacological Actions Researched for Insulin-like Growth Factor (IGF-1)
NameACCKFocus
Neuroprotective Agents47Locked
Apoptotic23Locked
Neurorestorative12Locked

View the Evidence:
5 Abstracts with Insulin-like Growth Factor (IGF-1) Research

[+]

Co-administration of IGF-I and glycosaminoglycans greatly delays motor neuron disease in the mouse model.Apr 01, 2002

Pubmed Data : J Neurochem. 2002 Apr;81(1):194-202. PMID: 12067233
Article Published Date : Apr 01, 2002
Study Type : Animal Study
Additional Links
Substances : Glycoaminoglycans, Insulin-like Growth Factor (IGF-1)
Diseases : Amyotrophic Lateral Sclerosis, Motor Neuron Disease
Pharmacological Actions : Neuroprotective Agents
[+]

Glycosaminoglycans boost insulin-like growth factor-I-promoted neuroprotection, indicating their potential value in motor neuron disease and peripheral neuropathies.Jan 01, 1999

Pubmed Data : Neuroscience. 1999;93(2):565-72. PMID: 10465440
Article Published Date : Jan 01, 1999
Study Type : Animal Study
Additional Links
Substances : Glycoaminoglycans, Insulin-like Growth Factor (IGF-1)
Diseases : Amyotrophic Lateral Sclerosis, Motor Neuron Disease, Peripheral Neuropathies
Pharmacological Actions : Apoptotic, Neuroprotective Agents
Additional Keywords : Drug Synergy
[+]

Glycosaminoglycans co-administration enhance insulin-like growth factor-I neuroprotective and neuroregenerative activity in traumatic and genetic models of motor neuron disease.Jul 01, 2000

Pubmed Data : Int J Dev Neurosci. 2000 Jul-Aug;18(4-5):339-46. PMID: 10817918
Article Published Date : Jul 01, 2000
Study Type : Animal Study
Additional Links
Substances : Glycoaminoglycans, Insulin-like Growth Factor (IGF-1)
Diseases : Amyotrophic Lateral Sclerosis, Motor Neuron Disease
Pharmacological Actions : Neuroprotective Agents, Neurorestorative
Additional Keywords : Natural Substance Synergy
[+]

IGF-1 improves fatigue resistance in the rat model of muscular dystrophy.Sep 01, 2004

Pubmed Data : Muscle Nerve. 2004 Sep;30(3):295-304. PMID: 15318340
Article Published Date : Sep 01, 2004
Study Type : Animal Study
Additional Links
Substances : Insulin-like Growth Factor (IGF-1)
Diseases : Muscular Dystrophies
[+]

IGF-I prevents glutamate-induced motor neuron programmed cell death.Jan 15, 2009

Pubmed Data : Food Chem Toxicol. 2009 Jan 15. PMID: 15193297
Article Published Date : Jan 15, 2009
Study Type : In Vitro Study
Additional Links
Substances : Insulin-like Growth Factor (IGF-1)
Diseases : Amyotrophic Lateral Sclerosis, Excitotoxicity
Pharmacological Actions : Apoptotic, Neuroprotective Agents

Copyright: rob3000 / 123RF Stock Photo

Insulin-like Growth Factor (IGF-1) Related Articles

Cancer & Sugar - Strategy for Selective Starvation of Cancer
Written by Dr. Mark Sircus
Whereas many within the mainstream medical community insist on promoting the belief that the link between certain types of food with an increased risk of cancer is "weak" or only "nominally significant," the scientific evidence continues to accumulate indicating sugar feeds cancer.

Print Options

Some features are currently member only features. If you are already a member, please login. Otherwise, click here to become a member.
  • Printer-friendly version

Connect with GreenMedInfo

This website is for information purposes only. By providing the information contained herein we are not diagnosing, treating, curing, mitigating, or preventing any type of disease or medical condition. Before beginning any type of natural, integrative or conventional treatment regimen, it is advisable to seek the advice of a licensed healthcare professional.

© Copyright 2008-2024 GreenMedInfo.com, Journal Articles copyright of original owners, MeSH copyright NLM.